Module 5 Quiz

A  the exclusion of electrolytes in certain measurements when pH, PCO2, and PO2 are elevated in arterial blood.

B  an underestimation of electrolyte concentration by indirect ISE methods when hyperproteinemia or hyperlipemia is present.

C  the exclusion of certain solids by increased activity of electrolytes when determining concentration of those solids.

D  an overestimation of electrolyte concentration by direct ISE measurements only when serum is used.

A  shift to the left.

B  remain unchanged.

C  shift to the right.

A  the PO2 of alveolar air.

B  the amount of total CO2 bound to hemoglobin.

C  the hemoglobin binding capacity of blood.

A  Sodium

B  Bicarbonate

C  Chloride

A  equivalents per liter

B  moles per liter

C  moles of osmotic substances per liter

D  moles of osmotic substances per kilogram

A  P50.

B  the half-life of oxygen.

C  oxygen dissociation.

D  oxygen saturation.

E  P50 is defined as the PO2 at which the hemoglobin of the blood is half saturated with O2.

A  Lp(a) does not contain any apo B-100.

B  Lp(a) interacts with ABCA1 in peripheral cells, such as the macrophages, to remove additional cholesterol.

C  Lp(a) contains a carbohydrate-rich protein called apo(a) that is covalently bound to apo B-100 through a disulfide linkage.

D  Lp(a) contains more core lipids, triglyceride, and cholesteryl ester and a smaller percent of protein.

A  HMG CoA reductase

B  Lecithin cholesterol acyltransferase

C  Cholesterol oxidase

D  Fatty acid hydrolase

A  prostaglandins.

B  sphingolipids.

C  terpenes.

D  acylglycerols.

A  there is no physical separation of HDL from the non-HDL fractions and HDL is selectively measured.

B  measurement of the rate of oxygen consumption occurs before removal of non-HDL lipoproteins.

C  precipitation of non-HDL lipoproteins must occur using polyanions.

D  enzymatic measurement of HDL occurs after non-HDL lipoproteins are removed by ultracentrifugation.

A  Genetic lack of LDL receptors on cell surfaces

B  Tangier disease

C  Insulin deficiency

D  Lipoprotein lipase deficiency

A  Exogenous

B  Intracellular-cholesterol transport

C  Endogenous

D  Reverse-cholesterol transport

A  It is a myocardial protein that regulates muscle contraction through interaction with myosin and actin.

B  CRP levels rise because of its presence in cardiac muscle and release upon muscle cell death.

C  It is an enzyme found in skeletal muscle and cardiac muscle and is released upon tissue injury.

D  It is an acute phase reactant plasma protein that rises in response to inflammation, which has been demonstrated to be associated with risk of future CHD.

A  Glucose oxidase method

B  Self-monitoring method

C  Hexokinase method

D  Polarographic method

A  Nicotinamide adenine dinucleotide and NADH

B  Glucose-6-phosphate and adenosine diphosphate (ADP)

C  6-Phosphogluconate and hydrogen ions

D  Glycosylated hemoglobin

A  glycogen; the liver

B  glycoprotein; cell membranes

C  starch; skeletal muscle

D  cellulose; vegetable fibers

A  Glucagon stimulates insulin release.

B  Glucagon inhibits gluconeogenesis.

C  Glucagon inhibits glycogenesis.

D  Glucagon stimulates gluconeogenesis.

A  glycogenesis.

B  glyconeogenesis.

C  glycolysis.

D  glycogenolysis.

A  glycogenolysis.

B  glycogenesis.

C  gluconeogenesis.

D  glycolysis.

A  Oral glucose tolerance test

B  Assessment of fasting blood glucose during a hypoglycemic episode

C  A 72-hour fast

D  Postprandial glucose analysis